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Home>Products>Injectable Anabolic Steroids>Lidocaine HCl Lidocaine Hydrochloride local anaesthetic drugs CAS 73-78-9

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  • Lidocaine HCl Lidocaine Hydrochloride local anaesthetic drugs CAS 73-78-9
  • Lidocaine HCl Lidocaine Hydrochloride local anaesthetic drugs CAS 73-78-9
  • Lidocaine HCl Lidocaine Hydrochloride local anaesthetic drugs CAS 73-78-9
  • Lidocaine HCl Lidocaine Hydrochloride local anaesthetic drugs CAS 73-78-9

Lidocaine HCl Lidocaine Hydrochloride local anaesthetic drugs CAS 73-78-9

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  • 73-78-9

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Wuhan Hezhong Biochemical Manufacturing Co., Ltd.

Wuhan Hezhong Biochemical Manufacturing Co., Ltd.China

  • Wuhan Hezhong Biochemical Manufacturing Co., Ltd.2020-07-10 09:46:19
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Product Details

Product Name: Lidocaine Color: White Assay: 99%
Packing: 25kg/cardboard Drum Grade: Pharmaceutical Grade Sample: Acceptalbe
Type: Plant Extract

Product Description



Lidocaine HCl Lidocaine Hydrochloride local anaesthetic drugs CAS 73-78-9

Quick Detail:

Product nameLidocaine Hydrochloride
Other nameLidocaine HCL
CAS73-78-9
MFC14H23ClN2O
MW;270.7982
Molecular structure
Assay99%
Appearance:White crystalline powder;
Usagesoluble in water or ethanol,dissolved in chloroform, insoluble in ether.
josiesteroids
Whatsapp:008618038186817


Description;

Lidocaine may be absorbed following topical administration to mucous membranes, its rate and extent of absorption varies depending upon concentration and total dose administered, the specific site of application and duration of exposure. In general, the rate of absorption of local anesthetic agents following topical application occurs most rapidly after intratracheal administration. Lidocaine is also well-absorbed from the gastrointestinal tract, but little intact drug may appear in the circulation because of biotransformation in the liver.

Lidocaine is metabolized rapidly by the liver, and metabolites and unchanged drug are excreted by the kidneys. Biotransformation includes oxidative N-dealkylation, ring hydroxylation, cleavage of the amide linkage, and conjugation. N-dealkylation, a major pathway of biotransformation, yields the metabolites mono-ethylglycinexylidide and glycinexylidide. The pharmacological / toxicological actions of these metabolites are similar to, but less potent than, those of lidocaine. Approximately 90% of lidocaine administered is excreted in the form of various metabolites, and less than 10% is excreted unchanged.

Specification;

ItemsSpecification
Appearancewhite or almost white crystalline powder
Identification1) Melting point 74-79 o C
2)IR
3)complies with the requirement
4) complies with the requirement
Appearance of solutionclear and colorless
Ph4.0-5.5
Related substanceImpurity A ≤0.01%
Single unspecified impurity ≤0.1%
Total impurities ≤0.5%
Heavy metals≤5ppm
Water5.5-7%
Sulphated ash≤0.1%
Assay99-101%(C14H23ClN2O, anhydrous substance)


Standard
USP
Item
INDEX
TEST
Appearance
A white, crystalline powder
A white, crystalline powder
Melting Point
74~79℃
75~77℃
Identification
<A> <B>
comply with the requirement
<A> <B>
Sulfate
Comply with the requirement
Comply with the requirement

Moisture

5.0~7.0%
6.4%
Sulphated Ash
≤0.1%
0.01%
Heavy Metals
≤20ppm
<20ppm
Assay
97.5~102.5%
100.1%


Applications;

Lidocaine crosses the blood-brain and placental barriers, presumably by passive diffusion.Studies of lidocaine metabolism following intravenous bolus injections have shown that the elimination half-life of this agent is typically 1.5 to 2 hours. Because of the rapid rate at which lidocaine is metabolized, any condition that affects liver function may alter lidocaine kinetics. The half-life may be prolonged two-fold or more in patients with liver dysfunction. Renal dysfunction does not affect lidocaine kinetics but may increase the accumulation of metabolites.


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